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Showing 1 to 12 of 292 entries
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Erratum to: The Mitogen-Activated Protein Kinase (MAPK) Signaling Pathway as a Discovery Target in Stroke.

Journal of molecular neuroscience : MN

Sun J, Nan G.
PMID: 27236649
J Mol Neurosci. 2016 Jul;59(3):430. doi: 10.1007/s12031-016-0767-y.

No abstract available.

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Sun J, Nan G. Erratum to: The Mitogen-Activated Protein Kinase (MAPK) Signaling Pathway as a Discovery Target in Stroke. J Mol Neurosci. 2016;59(3):430doi: 10.1007/s12031-016-0767-y.
Sun, J., & Nan, G. (2016). Erratum to: The Mitogen-Activated Protein Kinase (MAPK) Signaling Pathway as a Discovery Target in Stroke. Journal of molecular neuroscience : MN, 59(3), 430. https://doi.org/10.1007/s12031-016-0767-y
Sun, Jing, and Nan, Guangxian. "Erratum to: The Mitogen-Activated Protein Kinase (MAPK) Signaling Pathway as a Discovery Target in Stroke." Journal of molecular neuroscience : MN vol. 59,3 (2016): 430. doi: https://doi.org/10.1007/s12031-016-0767-y
Sun J, Nan G. Erratum to: The Mitogen-Activated Protein Kinase (MAPK) Signaling Pathway as a Discovery Target in Stroke. J Mol Neurosci. 2016 Jul;59(3):430. doi: 10.1007/s12031-016-0767-y. PMID: 27236649.
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RETRACTED ARTICLE: TN-2 Ameliorates Tunicamycin-Induced Mitochondria and Endoplasmic Reticulum Stress-Associated Apoptosis in Rat Dorsal Root Ganglion Neurons.

Journal of molecular neuroscience : MN

Deng Q, Xu C, Wang R, Zheng J, Li S, Jin Q, Yang Y.
PMID: 26076740
J Mol Neurosci. 2015 Oct;57(2):314. doi: 10.1007/s12031-015-0599-1. Epub 2015 Jun 16.

No abstract available.

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Deng Q, Xu C, Wang R, et al. RETRACTED ARTICLE: TN-2 Ameliorates Tunicamycin-Induced Mitochondria and Endoplasmic Reticulum Stress-Associated Apoptosis in Rat Dorsal Root Ganglion Neurons. J Mol Neurosci. 2015;57(2):314doi: 10.1007/s12031-015-0599-1.
Deng, Q., Xu, C., Wang, R., Zheng, J., Li, S., Jin, Q., & Yang, Y. (2015). RETRACTED ARTICLE: TN-2 Ameliorates Tunicamycin-Induced Mitochondria and Endoplasmic Reticulum Stress-Associated Apoptosis in Rat Dorsal Root Ganglion Neurons. Journal of molecular neuroscience : MN, 57(2), 314. https://doi.org/10.1007/s12031-015-0599-1
Deng, Qiukui, et al. "RETRACTED ARTICLE: TN-2 Ameliorates Tunicamycin-Induced Mitochondria and Endoplasmic Reticulum Stress-Associated Apoptosis in Rat Dorsal Root Ganglion Neurons." Journal of molecular neuroscience : MN vol. 57,2 (2015): 314. doi: https://doi.org/10.1007/s12031-015-0599-1
Deng Q, Xu C, Wang R, Zheng J, Li S, Jin Q, Yang Y. RETRACTED ARTICLE: TN-2 Ameliorates Tunicamycin-Induced Mitochondria and Endoplasmic Reticulum Stress-Associated Apoptosis in Rat Dorsal Root Ganglion Neurons. J Mol Neurosci. 2015 Oct;57(2):314. doi: 10.1007/s12031-015-0599-1. Epub 2015 Jun 16. PMID: 26076740.
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Retraction Note to: Nucleolin Promotes TGF-β Signaling Initiation via TGF-β Receptor I in Glioblastoma.

Journal of molecular neuroscience : MN

Lv S, Zhang J, Han M, Wang W, Zhang Y, Zhuang D, Shi R, Bian R, Yao C.
PMID: 28620837
J Mol Neurosci. 2017 Aug;62(3):430. doi: 10.1007/s12031-017-0935-8.

No abstract available.

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Lv S, Zhang J, Han M, et al. Retraction Note to: Nucleolin Promotes TGF-β Signaling Initiation via TGF-β Receptor I in Glioblastoma. J Mol Neurosci. 2017;62(3):430doi: 10.1007/s12031-017-0935-8.
Lv, S., Zhang, J., Han, M., Wang, W., Zhang, Y., Zhuang, D., Shi, R., Bian, R., & Yao, C. (2017). Retraction Note to: Nucleolin Promotes TGF-β Signaling Initiation via TGF-β Receptor I in Glioblastoma. Journal of molecular neuroscience : MN, 62(3), 430. https://doi.org/10.1007/s12031-017-0935-8
Lv, Shunzeng, et al. "Retraction Note to: Nucleolin Promotes TGF-β Signaling Initiation via TGF-β Receptor I in Glioblastoma." Journal of molecular neuroscience : MN vol. 62,3 (2017): 430. doi: https://doi.org/10.1007/s12031-017-0935-8
Lv S, Zhang J, Han M, Wang W, Zhang Y, Zhuang D, Shi R, Bian R, Yao C. Retraction Note to: Nucleolin Promotes TGF-β Signaling Initiation via TGF-β Receptor I in Glioblastoma. J Mol Neurosci. 2017 Aug;62(3):430. doi: 10.1007/s12031-017-0935-8. PMID: 28620837.
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Erratum to: The Clinical Utility of TIMP3 Expression in ACTH-Secreting Pituitary Tumor.

Journal of molecular neuroscience : MN

Sun B, Liu X, Yang Y, Dai C, Li Y, Jiao Y, Wei Z, Yao Y, Feng M, Bao X, Deng K, Wang N, Wang R.
PMID: 26894265
J Mol Neurosci. 2016 Apr;58(4):533-4. doi: 10.1007/s12031-016-0725-8.

No abstract available.

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Sun B, Liu X, Yang Y, et al. Erratum to: The Clinical Utility of TIMP3 Expression in ACTH-Secreting Pituitary Tumor. J Mol Neurosci. 2016;58(4):533-4doi: 10.1007/s12031-016-0725-8.
Sun, B., Liu, X., Yang, Y., Dai, C., Li, Y., Jiao, Y., Wei, Z., Yao, Y., Feng, M., Bao, X., Deng, K., Wang, N., & Wang, R. (2016). Erratum to: The Clinical Utility of TIMP3 Expression in ACTH-Secreting Pituitary Tumor. Journal of molecular neuroscience : MN, 58(4), 533-4. https://doi.org/10.1007/s12031-016-0725-8
Sun, Bowen, et al. "Erratum to: The Clinical Utility of TIMP3 Expression in ACTH-Secreting Pituitary Tumor." Journal of molecular neuroscience : MN vol. 58,4 (2016): 533-4. doi: https://doi.org/10.1007/s12031-016-0725-8
Sun B, Liu X, Yang Y, Dai C, Li Y, Jiao Y, Wei Z, Yao Y, Feng M, Bao X, Deng K, Wang N, Wang R. Erratum to: The Clinical Utility of TIMP3 Expression in ACTH-Secreting Pituitary Tumor. J Mol Neurosci. 2016 Apr;58(4):533-4. doi: 10.1007/s12031-016-0725-8. PMID: 26894265.
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RETRACTED ARTICLE: The Neuroprotective Effect of Histamine H4 Receptor Antagonist on Dopaminergic Neurons in Parkinson's Disease.

Journal of molecular neuroscience : MN

Cao X, Xiao H, Yang M, Chen X.
PMID: 30798471
J Mol Neurosci. 2019 Jun;68(2):319. doi: 10.1007/s12031-018-1242-8. Epub 2019 Feb 23.

No abstract available.

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Cao X, Xiao H, Yang M, et al. RETRACTED ARTICLE: The Neuroprotective Effect of Histamine H4 Receptor Antagonist on Dopaminergic Neurons in Parkinson's Disease. J Mol Neurosci. 2019;68(2):319doi: 10.1007/s12031-018-1242-8.
Cao, X., Xiao, H., Yang, M., & Chen, X. (2019). RETRACTED ARTICLE: The Neuroprotective Effect of Histamine H4 Receptor Antagonist on Dopaminergic Neurons in Parkinson's Disease. Journal of molecular neuroscience : MN, 68(2), 319. https://doi.org/10.1007/s12031-018-1242-8
Cao, Xu, et al. "RETRACTED ARTICLE: The Neuroprotective Effect of Histamine H4 Receptor Antagonist on Dopaminergic Neurons in Parkinson's Disease." Journal of molecular neuroscience : MN vol. 68,2 (2019): 319. doi: https://doi.org/10.1007/s12031-018-1242-8
Cao X, Xiao H, Yang M, Chen X. RETRACTED ARTICLE: The Neuroprotective Effect of Histamine H4 Receptor Antagonist on Dopaminergic Neurons in Parkinson's Disease. J Mol Neurosci. 2019 Jun;68(2):319. doi: 10.1007/s12031-018-1242-8. Epub 2019 Feb 23. PMID: 30798471.
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Analysis of Candidate Genes at the IBGC1 Locus Associated with Idiopathic Basal Ganglia Calcification ("Fahr" Disease').

Journal of molecular neuroscience : MN

Oliveira JRM, Sobrido MJ, Spiteri E, Hopfer S, Meroni G, Petek E, Baquero M, Geschwind DH.
PMID: 29349717
J Mol Neurosci. 2007 Oct;33(2):151-154. doi: 10.1007/s12031-007-0030-7. Epub 2007 Aug 01.

Basal ganglia calcification (striatopallidodentate calcifications) can be caused by several systemic and neurological disorders. Familial Idiopathic Basal Ganglia Calcification (IBGC, "Fahr" disease'), is characterized by basal ganglia and extrabasal ganglia calcifications, parkinsonism and neuropsychiatric symptoms. Because of an increased...

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Oliveira JRM, Sobrido MJ, Spiteri E, et al. Analysis of Candidate Genes at the IBGC1 Locus Associated with Idiopathic Basal Ganglia Calcification ("Fahr" Disease'). J Mol Neurosci. 2007;33(2):151-154doi: 10.1007/s12031-007-0030-7.
Oliveira, J. R. M., Sobrido, M. J., Spiteri, E., Hopfer, S., Meroni, G., Petek, E., Baquero, M., & Geschwind, D. H. (2007). Analysis of Candidate Genes at the IBGC1 Locus Associated with Idiopathic Basal Ganglia Calcification ("Fahr" Disease'). Journal of molecular neuroscience : MN, 33(2), 151-154. https://doi.org/10.1007/s12031-007-0030-7
Oliveira, J R M, et al. "Analysis of Candidate Genes at the IBGC1 Locus Associated with Idiopathic Basal Ganglia Calcification ("Fahr" Disease')." Journal of molecular neuroscience : MN vol. 33,2 (2007): 151-154. doi: https://doi.org/10.1007/s12031-007-0030-7
Oliveira JRM, Sobrido MJ, Spiteri E, Hopfer S, Meroni G, Petek E, Baquero M, Geschwind DH. Analysis of Candidate Genes at the IBGC1 Locus Associated with Idiopathic Basal Ganglia Calcification ("Fahr" Disease'). J Mol Neurosci. 2007 Oct;33(2):151-154. doi: 10.1007/s12031-007-0030-7. Epub 2007 Aug 01. PMID: 29349717.
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Correction to: the Cysteine (Cys) Residues Cys-6 and Cys-111 in Mutant Superoxide Dismutase 1 (SOD1) A4V Are Required for Induction of Endoplasmic Reticulum Stress in Amyotrophic Lateral Sclerosis.

Journal of molecular neuroscience : MN

Perri ER, Parakh S, Vidal M, Mehta P, Ma Y, Walker AK, Atkin JD.
PMID: 32529537
J Mol Neurosci. 2020 Sep;70(9):1369. doi: 10.1007/s12031-020-01617-5.

The original version of this article unfortunately contained an error in Fig. 3. The image shown for "C57S" was incorrect.

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Perri ER, Parakh S, Vidal M, et al. Correction to: the Cysteine (Cys) Residues Cys-6 and Cys-111 in Mutant Superoxide Dismutase 1 (SOD1) A4V Are Required for Induction of Endoplasmic Reticulum Stress in Amyotrophic Lateral Sclerosis. J Mol Neurosci. 2020;70(9):1369doi: 10.1007/s12031-020-01617-5.
Perri, E. R., Parakh, S., Vidal, M., Mehta, P., Ma, Y., Walker, A. K., & Atkin, J. D. (2020). Correction to: the Cysteine (Cys) Residues Cys-6 and Cys-111 in Mutant Superoxide Dismutase 1 (SOD1) A4V Are Required for Induction of Endoplasmic Reticulum Stress in Amyotrophic Lateral Sclerosis. Journal of molecular neuroscience : MN, 70(9), 1369. https://doi.org/10.1007/s12031-020-01617-5
Perri, Emma R, et al. "Correction to: the Cysteine (Cys) Residues Cys-6 and Cys-111 in Mutant Superoxide Dismutase 1 (SOD1) A4V Are Required for Induction of Endoplasmic Reticulum Stress in Amyotrophic Lateral Sclerosis." Journal of molecular neuroscience : MN vol. 70,9 (2020): 1369. doi: https://doi.org/10.1007/s12031-020-01617-5
Perri ER, Parakh S, Vidal M, Mehta P, Ma Y, Walker AK, Atkin JD. Correction to: the Cysteine (Cys) Residues Cys-6 and Cys-111 in Mutant Superoxide Dismutase 1 (SOD1) A4V Are Required for Induction of Endoplasmic Reticulum Stress in Amyotrophic Lateral Sclerosis. J Mol Neurosci. 2020 Sep;70(9):1369. doi: 10.1007/s12031-020-01617-5. PMID: 32529537.
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Correction to: Limb Remote Ischemic Preconditioning Reduces Repeated Ketamine Exposure-Induced Adverse Effects in the Developing Brain of Rats.

Journal of molecular neuroscience : MN

Liu Y, Li AQ, Ma W, Gao YB, Deng LQ, Zhang C, Meng JH.
PMID: 32096125
J Mol Neurosci. 2020 Apr;70(4):632-633. doi: 10.1007/s12031-020-01480-4.

The original version of this article unfortunately contained mistakes in Fig. 2c, d and Funding section.

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Liu Y, Li AQ, Ma W, et al. Correction to: Limb Remote Ischemic Preconditioning Reduces Repeated Ketamine Exposure-Induced Adverse Effects in the Developing Brain of Rats. J Mol Neurosci. 2020;70(4):632-633doi: 10.1007/s12031-020-01480-4.
Liu, Y., Li, A. Q., Ma, W., Gao, Y. B., Deng, L. Q., Zhang, C., & Meng, J. H. (2020). Correction to: Limb Remote Ischemic Preconditioning Reduces Repeated Ketamine Exposure-Induced Adverse Effects in the Developing Brain of Rats. Journal of molecular neuroscience : MN, 70(4), 632-633. https://doi.org/10.1007/s12031-020-01480-4
Liu, Ying, et al. "Correction to: Limb Remote Ischemic Preconditioning Reduces Repeated Ketamine Exposure-Induced Adverse Effects in the Developing Brain of Rats." Journal of molecular neuroscience : MN vol. 70,4 (2020): 632-633. doi: https://doi.org/10.1007/s12031-020-01480-4
Liu Y, Li AQ, Ma W, Gao YB, Deng LQ, Zhang C, Meng JH. Correction to: Limb Remote Ischemic Preconditioning Reduces Repeated Ketamine Exposure-Induced Adverse Effects in the Developing Brain of Rats. J Mol Neurosci. 2020 Apr;70(4):632-633. doi: 10.1007/s12031-020-01480-4. PMID: 32096125.
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Fetal Neuroprotective Mechanism of Maternal Magnesium Sulfate: Proteomic Analysis.

Journal of molecular neuroscience : MN

Reicher L, Dabaja H, Ginsberg Y, Khatib N, Fouks Y, Attali E, Ross MG, Weiner Z, Beloosesky R.
PMID: 34761370
J Mol Neurosci. 2021 Nov 10; doi: 10.1007/s12031-021-01939-y. Epub 2021 Nov 10.

Mg supplementation has been shown to protect preterm fetuses from white and gray matter damage, but the mechanism is unclear. The purpose of this study was to study the effect of maternal inflammation on the overall protein panel of...

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Reicher L, Dabaja H, Ginsberg Y, et al. Fetal Neuroprotective Mechanism of Maternal Magnesium Sulfate: Proteomic Analysis. J Mol Neurosci. 2021;doi: 10.1007/s12031-021-01939-y.
Reicher, L., Dabaja, H., Ginsberg, Y., Khatib, N., Fouks, Y., Attali, E., Ross, M. G., Weiner, Z., & Beloosesky, R. (2021). Fetal Neuroprotective Mechanism of Maternal Magnesium Sulfate: Proteomic Analysis. Journal of molecular neuroscience : MN, . https://doi.org/10.1007/s12031-021-01939-y
Reicher, Lee, et al. "Fetal Neuroprotective Mechanism of Maternal Magnesium Sulfate: Proteomic Analysis." Journal of molecular neuroscience : MN vol. (2021). doi: https://doi.org/10.1007/s12031-021-01939-y
Reicher L, Dabaja H, Ginsberg Y, Khatib N, Fouks Y, Attali E, Ross MG, Weiner Z, Beloosesky R. Fetal Neuroprotective Mechanism of Maternal Magnesium Sulfate: Proteomic Analysis. J Mol Neurosci. 2021 Nov 10; doi: 10.1007/s12031-021-01939-y. Epub 2021 Nov 10. PMID: 34761370.
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RETRACTED ARTICLE: Role of TLR2 and TLR4 Signaling in Parkinson's Disease: An Insight into Associated Therapeutic Potential.

Journal of molecular neuroscience : MN

Kaur G, Behl T, Sehgal A, Singh S, Sharma N, Kumar A, Arora S, Bungau S.
PMID: 33687621
J Mol Neurosci. 2021 Nov;71(11):2429. doi: 10.1007/s12031-021-01811-z. Epub 2021 Mar 09.

No abstract available.

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Kaur G, Behl T, Sehgal A, et al. RETRACTED ARTICLE: Role of TLR2 and TLR4 Signaling in Parkinson's Disease: An Insight into Associated Therapeutic Potential. J Mol Neurosci. 2021;71(11):2429doi: 10.1007/s12031-021-01811-z.
Kaur, G., Behl, T., Sehgal, A., Singh, S., Sharma, N., Kumar, A., Arora, S., & Bungau, S. (2021). RETRACTED ARTICLE: Role of TLR2 and TLR4 Signaling in Parkinson's Disease: An Insight into Associated Therapeutic Potential. Journal of molecular neuroscience : MN, 71(11), 2429. https://doi.org/10.1007/s12031-021-01811-z
Kaur, Gagandeep, et al. "RETRACTED ARTICLE: Role of TLR2 and TLR4 Signaling in Parkinson's Disease: An Insight into Associated Therapeutic Potential." Journal of molecular neuroscience : MN vol. 71,11 (2021): 2429. doi: https://doi.org/10.1007/s12031-021-01811-z
Kaur G, Behl T, Sehgal A, Singh S, Sharma N, Kumar A, Arora S, Bungau S. RETRACTED ARTICLE: Role of TLR2 and TLR4 Signaling in Parkinson's Disease: An Insight into Associated Therapeutic Potential. J Mol Neurosci. 2021 Nov;71(11):2429. doi: 10.1007/s12031-021-01811-z. Epub 2021 Mar 09. PMID: 33687621.
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Peripheral Blood Biomarkers CXCL12 and TNFRSF13C Associate with Cerebrospinal Fluid Biomarkers and Infiltrating Immune Cells in Alzheimer Disease.

Journal of molecular neuroscience : MN

Wu Q, Kong W, Wang S.
PMID: 33687622
J Mol Neurosci. 2021 Jul;71(7):1485-1494. doi: 10.1007/s12031-021-01809-7. Epub 2021 Mar 09.

Neuroinflammation-induced neurodegeneration and immune cell infiltration are two features of Alzheimer disease (AD). This study aimed to identify potential peripheral biomarkers that interact with cerebrospinal fluid (CSF) and infiltrating immune cells in AD. Blood and CSF data were downloaded...

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Wu Q, Kong W, Wang S. Peripheral Blood Biomarkers CXCL12 and TNFRSF13C Associate with Cerebrospinal Fluid Biomarkers and Infiltrating Immune Cells in Alzheimer Disease. J Mol Neurosci. 2021;71(7):1485-1494doi: 10.1007/s12031-021-01809-7.
Wu, Q., Kong, W., & Wang, S. (2021). Peripheral Blood Biomarkers CXCL12 and TNFRSF13C Associate with Cerebrospinal Fluid Biomarkers and Infiltrating Immune Cells in Alzheimer Disease. Journal of molecular neuroscience : MN, 71(7), 1485-1494. https://doi.org/10.1007/s12031-021-01809-7
Wu, Qianqian, et al. "Peripheral Blood Biomarkers CXCL12 and TNFRSF13C Associate with Cerebrospinal Fluid Biomarkers and Infiltrating Immune Cells in Alzheimer Disease." Journal of molecular neuroscience : MN vol. 71,7 (2021): 1485-1494. doi: https://doi.org/10.1007/s12031-021-01809-7
Wu Q, Kong W, Wang S. Peripheral Blood Biomarkers CXCL12 and TNFRSF13C Associate with Cerebrospinal Fluid Biomarkers and Infiltrating Immune Cells in Alzheimer Disease. J Mol Neurosci. 2021 Jul;71(7):1485-1494. doi: 10.1007/s12031-021-01809-7. Epub 2021 Mar 09. PMID: 33687622.
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Neurodevelopment and Genetic Evaluation of Sotos Syndrome Cases with a Novel Mutation: a Single-Center Experience.

Journal of molecular neuroscience : MN

Muhsin E, Basak G, Banu D, Alper G, Mustafa S.
PMID: 34386909
J Mol Neurosci. 2021 Aug 12; doi: 10.1007/s12031-021-01897-5. Epub 2021 Aug 12.

Sotos syndrome is a non-progressive neurological disease with overgrowing, increased bone age, and developmental retardation. The aim of this study is to evaluate the prenatal, natal, and postnatal clinical findings of patients with Sotos syndrome. Sixteen patients suspected to...

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Muhsin E, Basak G, Banu D, et al. Neurodevelopment and Genetic Evaluation of Sotos Syndrome Cases with a Novel Mutation: a Single-Center Experience. J Mol Neurosci. 2021;doi: 10.1007/s12031-021-01897-5.
Muhsin, E., Basak, G., Banu, D., Alper, G., & Mustafa, S. (2021). Neurodevelopment and Genetic Evaluation of Sotos Syndrome Cases with a Novel Mutation: a Single-Center Experience. Journal of molecular neuroscience : MN, . https://doi.org/10.1007/s12031-021-01897-5
Muhsin, Elmas, et al. "Neurodevelopment and Genetic Evaluation of Sotos Syndrome Cases with a Novel Mutation: a Single-Center Experience." Journal of molecular neuroscience : MN vol. (2021). doi: https://doi.org/10.1007/s12031-021-01897-5
Muhsin E, Basak G, Banu D, Alper G, Mustafa S. Neurodevelopment and Genetic Evaluation of Sotos Syndrome Cases with a Novel Mutation: a Single-Center Experience. J Mol Neurosci. 2021 Aug 12; doi: 10.1007/s12031-021-01897-5. Epub 2021 Aug 12. PMID: 34386909.
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Showing 1 to 12 of 292 entries